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Ranolazine treatment for myocardial infarction? Effects on the development of necrosis, left ventricular function and arrhythmia

Authors: Sharon L Hale|||Robert A Kloner

Journal: Cardiovascular drugs and therapy

Publication Type: Journal Article

Date: 2014

DOI: 10.1007/s10557-014-6548-3

ID: 25112450

Affiliations:

Affiliations

    The Heart Institute, Good Samaritan Hospital, 1225 Wilshire Blvd, Los Angeles, CA, 90017, USA, sharon.hale@netscape.com.|||

Abstract

Ranolazine, an inhibitor of the late current of the cardiac action potential (late I(Na)), is a well established clinical treatment for chronic angina. The late INa in cardiac myocytes also plays an important role in the pathophysiology of acute myocardial ischemia and reperfusion, and thus is a potential therapeutic target to ameliorate consequences of myocardial infarction. In experimental animal models, ranolazine has been shown to reduce myocardial infarct size, improve left ventricular function, decrease ischemia/reperfusion-induced arrhythmias and improve outcome in heart failure. Here we focus specifically on data from in vivo animal studies of myocardial ischemia and reperfusion.


Chemical List

    Acetanilides|||Cardiotonic Agents|||Piperazines|||Ranolazine